nik_s

Here's an example of a study if you're interested [1]!

Some observations:

- The full translation of the 6:22 minute video takes about 22 seconds (17x real time)

- It recognizes the language by default (and did a good job to recognize it was french audio)

- MIT License [3]!

- The quality of the transcription is good, but not perfect.

- The quality of the translation (if you don't consider transcription errors as a translation error) is generally very good.

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The transcription:

> Bonjour à tous, <error>j'suis</error> espère que vous allez bien, c''est ENTI. Et aujourd', <error>aujourd',</error> on se retrouve <error>un peu physique</error> pour parler de la termo dynamique. Vous ne vous inquiétez pas, ça va bien se passer. On va y aller ensemble, <error>être à par exemple,</error> je vous accompagne à travers une série de vidéos pour vous expliquer les principes de base en termo dynamique. Et bah, c''est parti, on va y aller tranquillement. Lidée, c''est vous puissiez comprendre la termo dynamique dans son ensemble. Donc, je vais vraiment prendre mon temps pour <error>couplisser</error> bien comprendre les notions,

The translation:

> Hello everyone, I hope you're doing well, it's NT and today we find ourselves a little physical to talk about the thermo dynamic. Don't worry, it's going well, we're going to go together and be the same. I'm going to accompany you through a series of videos to explain the basic principles in thermo dynamic. Well, let's go, <error>we're going to go quietly</error>. The idea is that you can understand the thermo dynamic <error>in sound together</error>. So I'm really going to take my time to understand the notions,

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All in all very happy that OpenAI is publishing their models. If Stable Diffusion is any guide, people will hack some crazy things with this.

It concludes that all fasting lengths are beneficial, and are likely going to result in a) reduction in weight and waist circumference, b) beneficial effects on blood lipids, regulation of sugar and other blood-related parameters, including lower blood sugars and higher ketone body levels, c) an increase in physical and emotional well-being and absence of hunger, d) a high probability of decrease of pre-existing health-complaints, e) very limited chance of side-effects.

Most NFTs are like a title to a house. Like a title to a house, you can: 1. Prove you own it; 2. Sell it to someone.

Unlike the title to a house: 1. The party certifying your ownership is not the government, but the consensus rules of a blockchain; 2. The thing you own is not physical, but digital in nature; 3. Your ownership does not prevent anyone from copying the file itself for their own uses.

Regarding your point that the object you own might not be consistent: because of the cost relative of storing data on a blockchain, the actual digital thing you own isn't usually stored on the blockchain, but a hash of that file (e.g. [1]). Unless hashing is broken, this is cryptographic proof that the object does remain consistent.

A cursory glance at Coinbase's USDC's market cap [1] shows that it too is growing at almost exactly the same pace as tether's [2]. I think most players in this market would agree that Coinbase, for all of it's failings, is unlikely to be planning an exit scam at this point.

It doesn't disprove the whole thesis, and some elements of it might have merit, but at least one element of it seems weak to me.

If I understand correctly, in order to achieve this:

  1. a nanotube is created that contains a chemical compound (SHP1i) that inhibits the the "don't eat me" (antiphagocytic) signal of plaque.
  2. These nanotubes are engineered to be taken up by white blood cells.
  3. White blood cells tend to be more present at areas of inflammation (such as plaque), which in turn increases the chance that the chemical compound mentioned above will be released in areas where plaque is present.
  4. This presence of nanotube-loaded white blood cells at areas of plaque will turn off the antiphagocytic signals of all cells in this area,
  5. which induces plaque (and all other antiphagocytic cells in the area) to be removed through traditional cellular activity.

  - How do the nanotubes bind to the white blood cells? Is there a specific technique to achieve this?
  - If I understand correctly, the chemical compound (SHP1i) removes antiphagocytic signals indiscriminately, which causes issues with toxicity of most therapies that attempt to use this technique without tightly controlling delivery (because organs might become a target of white blood cells). What happens however if a patient has other areas of inflammation? Does this exclude patients with auto-immune diseases from benefiting from these approaches, for example?