This is essentially creating an auto-immune disease in old age - by having the body target it's own cells. It's also not clear how the researchers prevented the immune cells from dying in the Thymus (who's function is specifically to prevent immune cells from being coded to attack cells in the body - it's like a UAT environment).
...but the bigger question would be - If this protein encoded a protein that identified cells which should always be eliminated in old age; why hasn't any mammal evolved to start destroying them?
It's more likely that there is some trade-off which we have not discovered yet.
...but the bigger question would be - If this protein encoded a protein that identified cells which should always be eliminated in old age; why hasn't any mammal evolved to start destroying them?
It's more likely that there is some trade-off which we have not discovered yet.