CFTR, being a chloride channel, most likely leads to diabetes as a consequence of its effects on the voltage-regulated insulin release in beta cells from glucose sensing. [1]
It strikes me as unlikely to be related to the salt-wasting that occurs from failure to reabsorb chloride in the epithelium.
It strikes me as unlikely to be related to the salt-wasting that occurs from failure to reabsorb chloride in the epithelium.
1 = https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4104438/